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Juliana soarez

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Since he had an exponential growth in his business, he had the need to invest in the digitalization of his business and in streamlining processes, making them more efficient.

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Backup Regularly With a sound and regular backup policy, the possibility of data loss is greatly reduced. Besides that, the information is […].

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Computer security is increasingly important to keep the business running. We have prepared some tips to improve […].

Plasticity endows T cells to be influenced by their microenvironment and respond to it accordingly. A specific microenvironment is defined by a variety of factors, including biological and chemical composition, cell-cell interactions, but also metabolic and mechanical cues.

CD4 T cells are key drivers of rheumatoid arthritis RA , the most common inflammatory rheumatic disease. There is an unmet need for therapies that precisely target the mechanisms underpinning CD4 T cell dysregulation rather than overall T cell activation or the end products, inflammatory cytokines.

In rheumatoid arthritis, elucidating how the joint microenvironment imprints dysregulated T cell inflammation, drives and possibly diversifies their pathological functions could broaden the potential therapeutic targets, extending beyond the successful blockade of end-stage inflammatory mediators.

Our research breaches immunology and clinical studies. We start with clinical data and tissue samples from patients and probe for the cellular and molecular mechanisms involved.

This research line focuses on defining the basis and consequences of pathogenic T cell populations in the development of Rheumatoid Arthritis.

Additionally, tissue explants will be used to elucidate the role of tissue microenvironment in driving joint chronic inflammation. The compounded contribution of host intrinsic factors, immunological niches and viral genetic diversity to HIV-1 pathogenesis remain largely unknown.

First, sex has been a neglected host intrinsic variable, yet women display faster HIV disease progression. Second, the immunological niche offered by follicular T Tfh cells allows for HIV-1 ongoing replication even in successfully treated patients.

Finally, most of current knowledge on HIV-1 pathogenicity is based on the study of the fairly infrequent subtype B. The mechanisms underlying HIV-female bias, the establishment of Tfh replicating reservoirs and how HIV-genetic diversity impinge on pathogenicity remain largely undefined.

We are addressing HIV-1 infection as a continuum of host intrinsic biological factors, immunological niches and HIV-1 genetic diversity.

HIV-1 is primarily an infection of lymphoid tissues. Anti-retroviral therapy ART has rendered HIV-1 infection a manageable illness for those with access to treatment, still it does not clear reservoirs where the virus persists.

To dissect cellular and molecular characteristics of the follicular reservoir we are using human follicular T cells isolated from human tonsils through fluorescence-activated cell sorting FACS and infect them ex vivo with HIV We are pursuing the identification, at the molecular level, the activation and metabolic pathways hijacked by HIV-1 through a combination of flow cytometry, metabolic assays and confocal analysis.

Women mount stronger immune responses and experience more severe HIV-1 pathogenesis. The precise mechanism mediating the sexual dimorphism in HIV-1 pathogenesis is not known, partly because sex has not been considered a biological variable.

Using human blood and tonsillar samples, we aim at identifying the molecular determinants underpinning the interplay between sex hormones and HIV-1 infection in order to identify potential pathways for intervention, and to rationally control for biological differences relevant to therapeutic efficacy outcomes.

However, distinct HIV-1 subtypes have different biological properties including disease progression rates and cellular tropism.

The impact of HIV genetic diversity can only be optimally undertaken in countries with high HIV genetic diversity through first-rate clinical registry.

In Angola, we are sequencing HIV-1 mutations found in newborn and mother samples and associating them to drug resistance. This study goal is to be able to adjust the ART regimen of these newborns and their mothers to their HIV-1 sequences and will improve the quality of life of Angolan children.

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At this stage when the world is going through a period of pandemic due to the coronavirus, there are many computer attacks that proliferate throughout the world.

But what types of […]. The world is going through an unprecedented phase. The coronavirus has radically changed the way we live and, consequently, the way we work.

Many companies are adopting homeoffice as a way to guarantee business inheritance. However, this work format can bring serious risks to the security of data, so it is essential to adopt all […].

Computer security is increasingly important to keep the business running. We have prepared some tips to improve […].

System failures can cause very serious problems, especially if they reach customers. Therefore, it is increasingly common to see companies embracing IT redundancy.

Redundancy is essential to ensure the high availability of systems, networks and data. Repeating critical […]. When disaster occurs, it is difficult to continue normal business operations.

The teams are stressed and the level of tension in the organization increases exponentially. In an era when data is so highly valued in companies, there is great awareness of the need to protect information even in the most extreme scenarios.

HIV-1 is primarily an infection of lymphoid tissues. Anti-retroviral therapy ART has rendered HIV-1 infection a manageable illness for those with access to treatment, still it does not clear reservoirs where the virus persists.

To dissect cellular and molecular characteristics of the follicular reservoir we are using human follicular T cells isolated from human tonsils through fluorescence-activated cell sorting FACS and infect them ex vivo with HIV We are pursuing the identification, at the molecular level, the activation and metabolic pathways hijacked by HIV-1 through a combination of flow cytometry, metabolic assays and confocal analysis.

Women mount stronger immune responses and experience more severe HIV-1 pathogenesis. The precise mechanism mediating the sexual dimorphism in HIV-1 pathogenesis is not known, partly because sex has not been considered a biological variable.

Using human blood and tonsillar samples, we aim at identifying the molecular determinants underpinning the interplay between sex hormones and HIV-1 infection in order to identify potential pathways for intervention, and to rationally control for biological differences relevant to therapeutic efficacy outcomes.

However, distinct HIV-1 subtypes have different biological properties including disease progression rates and cellular tropism.

The impact of HIV genetic diversity can only be optimally undertaken in countries with high HIV genetic diversity through first-rate clinical registry.

In Angola, we are sequencing HIV-1 mutations found in newborn and mother samples and associating them to drug resistance.

This study goal is to be able to adjust the ART regimen of these newborns and their mothers to their HIV-1 sequences and will improve the quality of life of Angolan children.

Cerqueira Supervisor: Helena Soares. Selected Publications. Team photos. We aim to elucidate T cell involvemente in protecttive imune resposes project 1 and in autoimmune diseases project 2 in Human samples.

For additional information, please contact helena. For enquiries on available positions contact Helena Soares helena. Please add a letter detailing the reasons you are applying, your CV, and contact information of 3 references.

Online ahead of print. Journal of Immunology. Frontiers in Immunology. Orchestrating cytoskeleton and intracellular vesicle traffic to build functional immunological synapses.

Immunological Reviews. A regulated vesicle fusion cascade generates signaling nanoterritories that control T-cell activation at the immunological synapse.

Journal of Experimental Medicine. Single-molecule super-resolution microscopy: deeper and faster.

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